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of the patient in the general anaesthesia arm may have influenced the results.

One limitation of our study is that the intervention and control groups were not randomized. This was due to the fact that the patients’ surgeon decided to allocate patients for the two methods.

There was no strict protocol for the anaesthesia of the intervention group in our study. There was one period of CO~2~ insufflation after induction but no insufflation after termination of the operation. In our hands, hypercapnia (PaCO~2~\>50 mm Hg) with mild to moderate pain is not associated with increased risk. However, we cannot exclude the possibility of PaCO~2~ levels being more than 50 mm Hg in some of the patients.

Assessment of QoR-40 {#Sec10}
——————–

All patients were assessed preoperatively and then on POD 1, 2, 3, and 7. The effect of the intervention was observed from the first day of surgery to POD 7. The results of QoR-40 were good and there was no difference between the two groups. One of the advantages of the preoperative patient satisfaction questionnaire is that it makes a comparison between different groups pre- and post-operatively easier \[[@CR11]\].

Comparison of QoR-40 with other studies {#Sec11}
—————————————

Some authors have used the QoR-40 questionnaire pre- and post-operatively and found a poor outcome, with large differences between the pre- and post-operative scores \[[@CR9]\]. Hence, the use of the pre-operative QoR-40 may be insufficient to measure patient satisfaction. In our case, the fact that scores were not different on POD 7 may be linked to the fact that post-operative pain was assessed pre-operatively and not after the patient was discharged from the hospital. It has been shown that the patients suffer the most pain on the first 3 post-operative days \[[@CR10]\]. The mid- to long-term (PODs 3–7) impact of anaesthesia-associated problems (e.g., post-operative pain) on patient satisfaction has not been studied much. One of the reasons could be that the QoR-40 questionnaire has not been used in long-term studies. In a large study, post-operative pain at POD 1 was found

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Relative testis size and the localisation of aromatase and testosterone were examined in golden lion tamarins. Testis size increases from 0.6 to 1.5 g in adult males but decreases in adult females. Aromatase appears to have a widespread distribution in the testis. Overexpression of aromatase is found in the lamina propria (interstitial cells), but it is not site-specific. Testicular aromatase appears during the early stages of spermatogenesis. Testosterone is found in Leydig cells and in epithelial cells of the seminiferous tubules at all stages. The peripheral zone of the seminiferous tubules shows intense aromatase immunoreactivity at all stages of development. Seminal vesicles contain a small amount of this enzyme.Horford and his Celtics were NBA playoff-tested after his fifth and fourth seasons in Boston, respectively. Now, heading into the 2017-18 season, he has been selected as part of the Cavaliers’ 2017-18 training camp in the hopes that he can help Cleveland become a championship contender.

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Apocynin (APO), a small molecule inhibitor of NADPH oxidase has been proved to exhibit anti-nociceptive activity in animal models of nociception. Currently, its underlying mechanism is poorly understood. The present study was designed to assess the anti-hyperalgesic effect of APO and to elucidate its potential mechanism of action in a rat model of formalin induced nociception. Adult male Wistar rats received formalin injection in the rat plantar surface of hind paw, with APO (12.5 or 25 mg/kg, i.p.) or vehicle treatment 20 min prior to formalin injection. Nociceptive behavior (mechanical allodynia and hyperalgesia), inflammatory cytokine level (IL-1β, TNF-α, and IL-10) and lipid peroxidation status (MDA and GSH) were measured 1h post formalin injection. APO dose-dependently attenuated nociceptive behavior of the formalin-induced hyperalgesia. APO produced anti-nociceptive effect and reduced the levels of plasma IL-1β and TNF-α in a dose-dependent manner. Furthermore, APO treatment decreased spinal IL-1β and TNF-α, increased spinal IL-10 expression. Additionally, APO treatment also decreased spinal MDA levels and enhanced spinal GSH levels in a dose-dependent manner. The administration of APO results in attenuated nociceptive behavior, decreased plasma IL-1β and TNF-α, attenuated spinal IL-1β and TNF